Gary Brecka

Wow, get stumped by many questions, you know, straight into it. So I, I'll, I'll tell what's more exciting than the treatments and the protocols, um, is the realisation that we are not as sick or diseased or as pathological as we think we are. Um, very often we are nutrient deficient. And I also believe that given the state of chronic disease, the pandemic of autoimmune conditions, uh, hormone imbalance, the age ranges that these are creeping down into, you know, uh, chronic disease, morbid obesity, type two diabetes, um, and this, this parabolic rise in autism, um, ADD, ADHD, OCD, manic depression, bipolar, um, you have to ask yourself, you know, is there something systemically wrong with society? Um, or is, is there something else that's underlying it? And what's really exciting to me, um, are some of the tests now for what I call the big four. Mold, mycotoxin, heavy metals, parasites, and viruses. Yeah. Um, you know, if you're listening to this podcast right now and you've had a strange myriad of symptoms that, uh, your doctors just can't figure out and they're telling you that it's either all in your head, um, or, uh, that you need to get stress out of your life, or you have one of these catch-all diagnoses like fibromyalgia, chronic fatigue syndrome, or if you're in any of the category of autoimmune diseases, uh, because autoimmune as a category is 90% idiopathic, meaning 90% of autoimmune conditions are of unknown origin. Yeah. Which I don't believe anything is of unknown origin in the human body. So when things don't show up on a lab and you know that you don't feel how you should, brain fog, weight gain, water retention, uh, poor focus and concentration, hormone imbalance, uh, poor sleep patterns, chronic fatigue, um, diffuse muscular pain. When you have any of those conditions, before you accept that you have a disease or a pathology, you should really look at the big four: mold, mycotoxin, heavy metal, parasite. And I believe if we would just take a step back and use those diagnostic tools to, for, look for these invaders, we, we'd find a lot of these. Um, now of course, I'm also really excited about, um, the convergence of artificial intelligence, of big data, and early detection. It's going to change everything. It's going to change everything. I believe if you're alive in five years from today, it will be your choice whether or not you want to live to 120 or 140.

I say five years. Yeah. Because the, the, the parabolic rise now in data collection and analytics, because you know, artificial intelligence can only take 700 trillion independent variables and create an actionable result. Um, but it, it can actually detect a lot of these conditions at what we're calling stage zero, right? Um, you know, detecting cancers at the circulating tumor cell stage, um, or the VOCC stage, which is what we would call pre-stage one. Um, you know, stage, stage zero. And and also looking at the early onset of a lot of these chronic conditions, soft blocking things like that before you have things show up on MRIs and imaging as a sclerosis or arterial sclerosis. You know, the care that and understanding that we have the brain, um, the science inside the cell that has gotten down, uh, to an organelle called the mitochondria, which is essentially the powerhouse of the cell. We know now that aging is a combination of two things. Um, immune fatigue, the progressive slow overwhelm of the immune system. Um, and I think the majority of people, uh, know that the immune system, it protects us from pathogens and invaders and bacteria and viruses and what have you, which it does. Yeah. Uh, but we're only now really realizing that the immune system, uh, about 65% of its function is to police ourselves. Yeah. Right? It's, it's to actually keep the, our entire system in check. And, you know, when we have an immune system that is busy fighting toxicity from our diet, from environmental toxins, from the, the sheer toxic load of just being in a modern society, um, it allows its normal operations to, uh, not be paid attention to. So cells called senescent cells begin to slip by. Um, these are, these are cells that are in everybody's blood. Red blood cells that can't carry oxygen, white blood cells that can't amount immune defense, platelets that can no longer carry growth factors. And when you get a proliferation of these types of useless cells that are taking up space and not allowing a healthy young cell to take its place and and, uh, do its role, uh, you, you start to advance, um, aging. You know, what's, what's really interesting is we age slowly in our early years and aging accelerates around age 40. Um, and it takes a parabolic spike at age 65. And so we begin to die faster the older that we get. So aging is not a linear, um, process. It's a curvilinear process. It's a, you know, looks like a hockey stick. And we have the capacity now to completely flatten that curve. So life extension is not the question. We certainly have the ability to extend life. Uh, extending healthspan is a really exciting thing. Um, you know, the number of healthy years that you have, um, you know, regenerating tissues with stem cell therapies, exosome therapies, natural killer cell therapies. So I would say if I had to pick a category, I'm really excited about the convergence of artificial intelligence, early detection, and big data. Um, you know, in the modality space, you know, I'm most excited about biologics, uh, stem cells, exosomes, natural killer cells. Um, and in the testing space, I'm most excited about the, the sensitivity of the new pathogenic testing.

I call, I call it HOAC as well, but I think the Americans call it...

Uh, I couldn't tell you. You know, please, please, please show. Hypothermic ozone, uh, uh, carbonic acid transdermal therapy. Um, and as I mentioned earlier, we've got patients coming into the clinic and after one session, it's, there's mold left in the base of the HOAC. It's incredible. And and they, and they their depression diminishes. They feel so much better. They just feel like new people.

Like in, in the United States, if you look at the PDR, the physician's, uh, desk reference, you'll find that we define depression, um, at least in, in America, um, as an inadequate supply of serotonin. So if you are low on serotonin, you are by definition depressed. So you would think if the definition of depression is low serotonin, that the fix would be to raise serotonin. Yeah. But that's actually not what we do. And we take people that are depressed, low on this neurotransmitter, and we put them on something called SSRIs, you know, selective serotonin reuptake inhibitors. So what these pharmaceuticals do is they ration what little serotonin you have. And so by definition, they never raise serotonin. So by definition, they never end depression. Um, I mean, I have dozens of clients that will come in to see me and it's just a band-aid.

I'll ask them, you know, how, how long have you been on an antidepressant? And they'll say, you know, 15, 18 years. Um, my first question is always, you know, when did you think it was going to kick in?

Um, but if we just take a step further and we say, okay, well, if, if I lack adequate supply of the neurotransmitter serotonin, um, then I can't reach the emotional states, the moods that require serotonin, right? Because if you asked me what is a mood, what is an emotional state, um, I would tell you that it is a collection of neurotransmitters in many cases bound to oxygen. Um, so in the absence of oxygen, you can't reach elevated emotional states. That's why nobody, no human being has ever woken up laughing. It's very easy to wake up angry. Those states don't require oxygen. But elevated emotional tears, passion, elation, joy, arousal, libido, those all require the presence of of oxygen in the brain. But, you know, if we, we just asked ourselves, well, where is serotonin made? Well, it's, it's actually made right here in the gut. You know, 90% of the serotonin in your body is right here. So if you don't have it here, you can't have it here. And so depression rarely begins in our outside environment. It almost always begins in our inside environment. And that's not to say that there are not valid reasons why people can be depressed. You know, loss of a loved one, financial crisis, you there are, there are life events that, you know, are plenty reason to be, um, depressed, but those should be temporary and fleeting. If you've struggled on and off, you know, throughout your lifetime with depression or anxiety or attention deficit disorder or, or any kind of mood disorder for that, that matter, um, you know, you really need to take a look at, um, how your body is methylating, how well you're converting nutrients. You know, if we went into the factory that makes serotonin, which is in the gut, um, and we said, "How do we make this neurotransmitter serotonin?" Well, we take an amino acid called tryptophan, we methylate it into the neurotransmitter serotonin, travels up the pathway called the vagus nerve, enters the brain and it begins to create mood and emotion. Same with dopamine. You know, we take phenylalanine, tyrosine, we methylate that into the neurotransmitter dopamine, the main driver of mood. We methylate serotonin, the main driver of behavior. So our mood and our behavior are heavily driven by neurotransmitters that are methylated, that are created in factories in our gut. And very often this factory is turned off or it's dysfunctional. Um, and it's a lot easier to turn back on than I think a lot of modern medicine, uh, fallacies about.

Yes. Um, but those are, uh, magnesium is a big one. Copper is rarely a deficiency, you know, related to that pathway, but the form of of of B12, B12 is a metal. A lot of people think it's just a vitamin. It's a vitamin, but it's a, it's a light metal. It's the cobalt metal. Um, when you put it into the body, it's called cobalamin. Um, when you don't have it in the methylated version, methylcobalamin, because the body will convert other forms of vitamin B12 into the methylated form, um, in order to use it, then you need to supplement with these things. And very often, you know, human beings are not as sick or diseased or as pathological as we think we are. We're nutrient deficient. Yeah. You know, we, we subscribe to this, uh, so, uh, wholeheartedly in, in plant physiology. You know, if you had a, a palm tree rotting in your, uh, you know, at your house, let's say you had a leaf that was rotting in a palm tree and and you called a true arborist, a true botanist out to your house, they wouldn't even touch the leaf, right? They would core test the soil. Yeah. And they would say, "You know what, Nick, there's no nitrogen in the soil." And they would add nitrogen to the soil and the leaf would heal. Human beings are no different. If you look at the vast array of what we call methylation, how the body takes, um, compounds that it can't use and converts them into the usable form and you were to fix those nutrient deficiencies, you would see true magic happen in the human body. Um, but in any case, I want to go back because I, I, I started by saying there are two main, uh, reasons for, for aging. You know, one, uh, you know, essentially being this immuno fatigue theory and the other is, you know, a slow progressive decline in the function of our mitochondria, you know, the powerhouse of the cell. All of the really good powerful anti-aging research, um, is now inside of the cell. Um, you know, we've gone through the cell wall, through the cytoplasm and we're into the mitochondria and we're realizing without the fuel to power here, power cellular metabolism, sort of nothing else matters. And a lot of the conditions that we are affected by and the pathogens that attack us, attack us by disabling our mitochondria. Um, and a lot of the toxic soup that we bathe our cellular biology in just with, you know, an highly processed diet.

Um, you know, these, these, these are actually, you know, affecting our mitochondria. And we know now how to rebuild a cell from the mitochondria out. And that's really exciting, uh, for humanity. Um, because I think we, anyone listening to this right now, um, is in the lifetime where you will see, uh, the end of cancer. At least the end of, um, cancer mortality. Um, people still get cancer. Um, but it's, it's, it's a really exciting time right now to be alive. Um, the pace of innovation is just, uh, mind-numbing right now. And I, I think humanity is going to be a big benefactor of this.

Oh my gosh, yes. I'm a huge, huge fan of peptides. I'm glad you said that. Yeah, peptides, um, for the most part, uh, the majority if not nearly all of peptides are essentially amino acid sequences, right? And when you, when you put certain, put certain chains of amino acids, very specific chains of amino acids into the body, um, they behave in a very unique way. Um, the exciting thing about peptides is because they are amino acids, um, they fall into the category of something called metabolites, which means that the body recognizes them, the body can recognize them, it can break them down, it can, can get rid of the waste. Um, whereas with a chemical or a synthetic or a pharmaceutical, um, the body doesn't have enzymes. It doesn't have waste elimination pathways. It doesn't have, uh, the chemical messengers to not only read but to also rid the body of the byproducts of chemicals, synthetics, pharmaceuticals.

And so when you use a peptide, you, you rarely desensitize to these types of of, uh, whatever you want to call them, biologics, because the body can recognize them, break them down, and get rid of them. It's the same way that we don't desensitize to food. You could eat 1500 calories a day for the rest of your life. It's not like when you're 20 years old, you eat 1500 calories and when you're 50, you eat 4,000 calories. You don't desensitize to food because the body can metabolize it and it can break it completely down and get rid of the waste. And so there are peptides that, uh, can influence our pituitary to return to a more youthful secretion of growth hormone. You know, GHRPS, GHRH is the growth hormone releasing hormones. Uh, there are ones that counteract anxiety. You have anti-anxiolytics. There are peptides now that show real promise for lengthening telomeres like epitalon, um, that are telomerase peptides that actually make the enzyme telomerase, which actually can recap our chromosomes, um, which is, you know, another one of the markers of of your biological age versus your chronological age. Um, and so it's, it's a really, really exciting field. I'm a big fan of peptides. I use a lot of peptides myself. Um, you can accelerate wound healing, BPC-157, TB5000, uh, you know, so they, they, they run the gamut. You have to be cautious about where you source them. You know, good compounders will not source their raw materials, uh, from China. Most do. Um, and if you have good sterility, stability and potency and and good manufacturing practices, I mean, peptides, I think, uh, would be as common as a multivitamin in five years.

Wow.

No question. Um, so, uh, thymosin alpha um or thymosin alpha 1 uh, is an incredible peptide for boosting and strengthening the immune system. And remember this theory of immuno fatigue is that our immune system eventually gets run down and can't take care of the house any longer. You know, you get really busy at your job and trash starts to build up at your house. Your house gets messier and messier and messier. The process of cellular metabolism is a very um is a very dirty process from a metabolic standpoint. And when the body is not capable of eliminating cellular waste, I'm not talking about stool and urine, the inflammatory factors and interleukins, cytokines and histamines and other um pro-inflammatory factors, you know, uh oxidative free radicals, then um the immune system is is busy doing other things and this is when disease and pathology set in. So if you're over the age of 40, you know, thymosin alpha is a peptide that you really ought to consider. I'm also an enormous fan of BPC-157 and TB5000. These are, these are wound healing peptides. What essentially they do is they capture the body's ability to heal itself. Um, so let's say you're walking down the street and you step off of a curb and you tear your right ligament in your ankle. Um, you know how does the body know to heal the right ligament and leave the left ligament alone? Well, the reason why it knows that is because when you damage tissue, let's say you take a ligament and you create a tear, uh, you damage a cell called a fibroblast and it and it begins to signal into the bloodstream, right? It begins to say, "I'm hurt, I'm hurt." So it sends these certain inflammatory factors um, in, into the bloodstream. And then a platelet, uh, which is cruising by in your bloodstream, which carries growth factors inside of it. It hears that signal and it bursts and it drops off growth factors. We've known for for well over a decade now that you can take platelets out of yourself, you know, platelet-rich plasma. You can spin your own blood down, you can concentrate your, your, uh, platelets, your plasma and you can inject it into the site of injury. Right? So if you have a torn rotator cuff, I'm not talking about a complete separation...

You would actually inject it where the injury actually is. Not, not BPC-157, but I'm talking about PRP, platelet-rich. Yes.

They, um, they try to, uh, you know, through the different types of centrifugation, they'll get down to the platelets and then essentially they will inject your own platelets from your own body. They will just concentrate them and inject them into the site of injury, knees, hips, shoulders.

Yeah, that's exactly the point that I'm making. So BPC-157 what it would do if you had a torn ligament is it would find that site of injury and it would amplify that signal. So instead of whispering into the bloodstream, it's screaming into the bloodstream. So as a as a pool of platelets are going by that site of injury in your blood and they hear that signal, they begin to burst in mass and migrate growth factors to that site. And so you're just accelerating your own capacity to heal, you know, from injury and not even necessarily from injury, but just from tissue damage, you know? Um, if you look at the single cell layer that lines the luminal wall of our gut, you know, there's, there's a single cell layer separates all of us, our inside environment from our outside environment. You know, the inside of your intestinal tract is contiguous with the side of your cheek. And so we think about our you know digestive tract and our intestines and our colon as being, you know, inside the body. They're not. They're external to the body. They just happen to run through the body. And so this single cell layer that's on the luminal wall of your gut is all that separates your inside environment from your outside environment. And it's very susceptible, uh, to all kinds of things, but it's very susceptible to something called leaky gut where you get tears um or you get breaks um in these tight junctions of the gut and then contents from inside your gut begin to migrate um into your bloodstream through the luminal wall of the gut. It calls the immune system to that site and wreaks havoc in your bloodstream and elsewhere in your body. BPC-157 is very good at healing and sealing the gut. In fact, it's a gastric pentadecapeptide, so it's synthesized from gastric juice. Um, very, very, very few side effects. There have been millions of prescriptions written for BPC-157. Um, has an extraordinarily good safety profile.

I, I'm curious why they would have such a bad reputation. Other, other, you know, here pharma has waged war against them, so I hear. So the FD, you know, the FDA pulled a lot of peptides from the market. You can still get them in your nutritional grade, but I think under this current administration, you're going to see those lanes widen again.

Yeah, I'm very involved in his movement, so I hope to be a small part of helping that happen here. That's great.

Well, so obviously the best way to get Vitamin D3 is from the sun. You know, we've been taught for decades to fear the sun. Uh, the sun is not our enemy. In high doses, just like anything else, you know, it can be very harmful. Um, if you're afraid of getting sunlight and and and burning, just um, you know, go in the sun during first light, the first 45 to 60 minutes of the day. There are no UVA or UVB rays. So none of the damaging rays that um cause, you know, uh tissue cell damage, only beneficial rays from the sun, the, the very healthy blue rays that do generate Vitamin D3. Um, it's one of the most prolific deficiencies in the world. Um, you know, Vitamin D3 was the deficiency was the second leading cause of morbidity in COVID.

Second leading cause of morbidity in COVID. The first was morbid obesity and and type two diabetes combined. Um, and the second was, uh, clinical deficiency in Vitamin D3. So, you know, an optimal range for Vitamin D3 is between 60 and 80 nanograms per deciliter. Um, and remember, Vitamin D3, cholecalciferol, um, is the only vitamin that human beings make on our own. So just imagine if I pulled your blood, the blood of anybody listening to this podcast, um, there would be hundreds of vitamins in their bloodstream. You're only capable of making one. You don't need to eat. You don't need to drink. You just need to expose your skin to sunlight and have cholesterol in your bloodstream. That's it. To make Vitamin D3. So think of how important something must be to human function if it's the only vitamin that we generate on our own. And if you really, I don't have you, we don't have time to go into all the mechanisms of Vitamin D3, but it acts like a vitamin. It acts like a hormone. Um, it's, it's, it's involved in calcium transport. It can actually take transport calcium around the bloodstream. It can deliver it to the bones or it can deliver it to the arterial wall if you don't have Vitamin K2 with it. It's, it's a phenomenal, um, phenomenal element. It really is. And, uh, yes, I do believe very strongly in Vitamin D3 supplementation. Um, remember Vitamin D is in the class of fat soluble vitamins. So A, D, E and K. Um, I remember in grad school my my professor used to say, "Put your vitamins in the attic," and ADK to to help us remember that those were the four fat soluble vitamins. And so when you take Vitamin D3, it will be useless if you don't actually have some fat. MCT oil, olive oil, some fat in your food to to help emulsify it and help transport. Um, and, uh, so it's important to take Vitamin D3 with food. Um, I would say a minimum 5,000 IUs of Vitamin D3 daily with...

Nothing. If you went out for 40 minutes in the sun, 30 minutes in the sun, with a pair of shorts or a woman in a bikini and laid 15 minutes on one side, 15 minutes on the other side, you would make endogenously about 25,000 IUs of vitamin, Vitamin D3. And so that's one of the best ways to get it. But, um, but supplementing with it exogenously and you can, you can test this on a lab. It's very easy. It's called, uh, it's called Vitamin D25 hydroxy, uh, is what it's called on a lab panel. But when you, when you look at your Vitamin D3 level, um, you want this to be between 60 and 80 nanograms per deciliter. Most people, um, the low end of the range is 30. Most people's Vitamin D3 is in the teens unless they're getting regular sunlight. Yeah. Um, this is critical for your immune system, for bone health, um, for calcium transport, to keep calcium out of the arterial wall and deposited into your bones is probably the single most important element, um, in the manufacture of of hormones. You know, Vitamin D3, DHEA, and then the production of nearly every hormone in the human body. And so, you know, this is getting back to my whole theme that, you know, we are very, you know, nutrient deficiencies lead to the expression of some of the most common conditions that we suffer from in, you know, mankind. So if your Vitamin D3 level is not between 60 and 80 nanograms per deciliter, um, absolutely supplementing with a good clean cholecalciferol Vitamin D3 with K2, make sure that you take one with K2 at the same time, um, with your first meal of the day can be a game changer.

Um, it's not too much if you're that deficient. Um, but between five and 10,000 is, you know, we, we, we've supplemented hundreds of thousands of patients with up to 10,000 IUs of Vitamin D3 a day. And unless they're getting, um, regular sun exposure, you'll see that their D3 doesn't get above 80 nanograms per deciliter. So those I would consider to be very safe ranges.

Well, they're the second most profitable pharmaceutical in...

Um, you know, uh, you know, very interestingly, if we just took a step back for a second and talked about what cholesterol is and what it's not. Cholesterol is is is not a fuel source. Your body cannot use cholesterol for energy. So then what is it, right? And and and just so people know, only 15% of the cholesterol in your bloodstream comes from your diet. 85% of the cholesterol in your blood is manufactured by the liver. So if you want to lower your cholesterol, you have to lower the liver's production of cholesterol. And if you want to lower the liver's production of cholesterol non-pharmacologically, you have to control what goes in the front door of the liver because it will decide what goes out the back door. And so high glycemic carbohydrates, um, um, especially, you know, refined carbohydrates are very good at raising your cholesterol. Yeah. So cholesterol is not a fuel source. It's a construction material. So we use it to build every cell wall, every cell membrane. Um, we use it to make Vitamin D3. Vitamin D3 is made from cholesterol. Um, nearly every hormone in the human body is made from cholesterol. So it's a very necessary construction material. More than 60% of your brain's weight is cholesterol. And cholesterol gets blamed for a lot of crimes that it doesn't commit. Um, it is very often at the scene of a crime, but it is rarely the one that pulled the trigger. Yeah. It's, you know, cholesterol is, is, um, I liken it to a fireman. You know, when, when you have a fire, the fireman shows up to put the fire out. If you don't have a fire at your house, I mean, I'm sitting in my house now. The likelihood of a fireman walking through my door is zero unless this place caught on fire. So it is called to the site of inflammation and it's called to the site of inflammation because usually because there is damage, um, there's damage to the arterial wall or to the vascular wall, what's called the endothelium. And if there weren't damage, cholesterol would not be called to that site to repair that damage. And so cholesterol gets blamed for a lot of crimes that it doesn't commit. Cholesterol amongst other things, is also a fat transport molecule. And so, um, just imagine that cholesterol was a tennis ball. Uh, the fuzzy yellow coating would be a triglyceride, right? So fat is carried around the bloodstream on the surface of cholesterol. So if you just took a cholesterol reading in your blood, for example, let's say that, uh, your cholesterol was 130 nanograms per deciliter. Um, your doctor's going to tell you that that's high. It's over 99. So you need to be on a statin. But what matters more than the amount of cholesterol is the size of the cholesterol molecule. Because large puffy balls of cholesterol in your bloodstream are very healthy. In fact, they are markers for longevity. Small BBs of cholesterol are markers for cardiovascular disease. And what is the difference? Well, if you, uh, if I take you back to high school geometry for a second, um, if you remember that as the size of a sphere gets smaller, its surface area to volume ratio goes up. So why that's important is let's say that we had two basketballs of cholesterol sitting on our desk. Okay? So they would be covered in fat, right? If you added more fat, triglyceride to the bloodstream, um, then and you kept the cholesterol the same, those two basketballs would have to become four softballs. If you added more fat, they would become eight baseballs. If you continued to add fat, they would become 16 golf balls. And if you still continued to add fat, they would become 32 little BBs. Now those two basketballs and those 32 little BBs are the same volume, the same nanograms per deciliter. You never change the amount of cholesterol. Although the basketballs are a marker for longevity and the BBs are a marker for cardiovascular disease. So what determines the size of cholesterol and its lethality? The level of triglyceride. So the higher your triglyceride to cholesterol ratio, the higher the risk for cardiovascular disease. Yeah. There's virtually zero evidence anywhere in the published clinical literature um linking elevated levels of LDL cholesterol on its own, just LDL cholesterol on its own to cardiovascular disease. You have to have a corresponding increase in triglyceride, which most people don't have. Now there are, if you look at a full cholesterol profile, you should really be looking at something called lipo little a and lipo b. These have a genetic component. Um, and uh there is no pharmaceutical intervention for these and it's it's important to be extra vigilant on um taking care of the rest of your metabolism, whole food diet, exercising, trying to keep inflammatory markers low, controlling your insulin. But for the majority of the population, um, how do we solve for this triglyceride to cholesterol ratio? You solve for the amount of sugar. You solve for insulin and blood sugar. I promise you, insulin is the root of all evil. The Bible says it's the love of money. Um, but I think that insulin is the root of all evil. Um, because, you know, as we become insulin resistant and we oxidize LDL cholesterol, now you have a dangerous particle in your body. Cholesterol itself is not the enemy.

Um, so first of all, I would, I would immediately engage in dietary and lifestyle changes. Um, you know, we, we, we, we've gotten away from thinking that we can control our own risk factors. Um, and, you know, when I was in the mortality space, uh, we had one of the largest population databases in the world for, um, uh, for mortality, 370 plus million lives. And we knew the day, date, time, location and cause of death for all of these. Um, and what we realized, and and the reason why I'm in this industry and not in that industry any longer, what we realized was that the reason why the majority of people are not living longer, healthier, happier, more fulfilling lives are for what we call modifiable risk factors. Right? So the question is, what got you to the point where you have this atherosclerotic plaque or these arterial sclerosis? Um, and generally you will find is a combination of things. Um, number one, insulin resistance. That is a the biggest villain. Um, hyperinsulinemia, high levels of insulin, high levels of blood sugar. Um, when you actually go and get a lab done, you should be looking at your glycemic profile, which is essentially three markers: your glucose, your hemoglobin A1C, which is the three-month average of your blood sugar, and your insulin. Because as your insulin becomes, um, less effective, which is called insulin resistance, the amount of insulin in your bloodstream begins to rise. And this high level of insulin, um, causes damage to the endothelium, the lining of the arteries. And so I would, um, I would, if you weren't already, immediately switch to a whole food diet. And by whole foods, I mean just foods in their natural form. Get as close to the soil as you can. Dogmatic dieting is not the savior of humanity. If you actually look at the longevity research or you have any familiarity with the blue zone research, you'll see that there's no continuity between diets. In other words, it wasn't keto, carnivore, pescatarian, vegan, vegetarian, whole, I mean, uh, uh, you know, Mediterranean. It wasn't a specific diet that dramatically extended life. The continuity between all of these diets, you know, if you go to Sardinia, one of the highest carbohydrate consumptions in the world, one of the longest life expectancies. You go to the Mediterranean, had one of the highest oil and fatty fish, you know, uh, consumptions and ultra long life expectancies. You go to Singapore, it's the highest meat per capita consumption per capita in the world and very long life expectancy. And then the French are kind of screwing up the whole model because they're drinking wine, cigarettes, eating cheese and they're living forever. So screw the French.

But but the point is that, um, you know, all of these diets were completely devoid of highly processed foods. So they were all whole food diets. So if you read ever read, uh, uh, Blue Zone Research or the Longevity Diet, um, book by, um, Dr. Valter Longo, um, it's not all meat, all fish, all olive oil, um, or any one dogmatic diet. It's putting whole foods into the body. And that's the first thing. Um, and and when you put whole foods into the body, something really magical happens. Um, you begin to secrete GLP-1s, right? GLP is the is the hormone that, um, you know, Ozempic and Mounjaro and all of these GLP-1 um agonists are mimicking. But this is a hormone that we make in our gut that responds to satiety, satiation, which is a direct result of nutrient density. So when we eat non-nutrient-dense foods, our brain continues to make us hungry so that we crave the intake of more foods in an attempt to get the nutrients that we need. Our brain's very smart. Um, you know, you could put six boxes or bags of Oreos on the counter and you could eat your way through all six bags, but you couldn't eat six avocados, right? You couldn't eat your way through that. The satiation, the GLP that you would release would be so high. The leptin, ghrelin balance would be so out of balance that you would start to feel physically sick even if you weren't physically full. And so the body has a a way of of telling us when when we've eaten. Our ancestors didn't count calories. They didn't know what macros were and micros were. They ate whole foods and they ate until they were satiated, right? Macro counting and and intermittent fasting. These are all, you know, the pandemic of the new age. You know, ancestrally when we actually had longer life expectancies than we do now, we ate to satiety. So the first thing I would do is switch to a whole food diet. And the second thing is I would do everything I could to increase my movement. Um, and, you know, that was a non-negotiable in in in in Blue Zone research was mobility until later in life. Um, as was sense of purpose and and community.

Yeah. Um, and I'm, you know, I'm going to be 55, you know, by the time I take it to Australia. So that's great. But but I feel amazing, you know, and and I, I exercise to not injure myself. I don't exercise to impress anyone. I'm, I'm working out for for longevity. Um, I feel amazing. I feel better now than I did when I was in my 20s. Um, but, uh, so, so just back to this plaquing for a moment. Uh, there's a compound called Plaquex that you can take.

Um, that you can take that actually can disrupt the fibrinogen bonding.

Plaquex. You can get it in an IV form and you can also get it in an oral form. Um, what this does is it disrupts fibrinogen bonding. Um, fibrinogen is what sort of holds these plaques together. Um, and if you take the, if you disrupt the fibrinogen bonding, you can take it from a waxy substance, um, to an oil, right? You don't want to actually crack plaques off. That can become an embolism. You, you, if you change the morphology of the, of the plaque from a, uh, from a wax to an oil, um, it seeps back into the bloodstream. Um, so I, I would be trying to control for my blood sugar, um, doing everything I could to lower my triglycerides, which will dramatically diminish your cardiovascular risk. I would look at markers of inflammation, specifically something called C-reactive protein, HSCP, and I would be looking at something called homocysteine. Um, homocysteine is is another silent killer and it and it flies by um detection. It's very, very easy to treat. Homocysteine is an amino acid that's in everybody listening to this podcast's blood right now. Um, and as your, if, if you have a genetic predisposition, for example, if you have the MTHFR gene mutation, which we affectionately call the [expletive] gene...

Whatever. So, uh, it's not the [expletive] gene. It stands for methylenetetrahydrofolate reductase. But what it, uh, what this gene does is it takes folic acid and folate and it, it methylates it into the form that the body can use called methylfolate. Now if you have this gene mutation, which roughly half the population has, um, then you, you, you have an impaired ability to make this conversion, which means you have a deficiency in methylfolate. When you have a deficiency in methylfolate, which is very easy to supplement with, the downstream consequences of that are that you have impaired homocysteine metabolism. Now homocysteine is a normal amino acid in everybody's blood. When it rises out of about the single digits, what happens when homocysteine rises is when it's cruising by the inside lining of the artery, it irritates the artery. And when you irritate an artery, it will clamp down. And if you make the pipes smaller in a fixed system, the pressure goes up.

There's so many people walking around right now with high blood pressure. And the type of high blood pressure they have is idiopathic hypertension, hypertension of an unknown origin. And they've had a full heart exam. They've had an EKG, normal EEG, normal heart sounds, normal lung sounds. They've had a dye contrast study, normal cardiac catheterization, normal CT scan, normal. They can't find anything wrong with the heart, but the pressure is still up. Well, you have to remember that we have 63,000 miles of blood vessel in our body. It doesn't take much arterial narrowing to drive that pressure up. Any plumber can tell you that if you want to double the pressure, cut the size of the pipes in half. So when you shrink the pipes, the...

Pressure rises. And very often your pipes are constricted because of this hyperhomocysteinemia, an elevated amino acid in the blood. Um, this is relatively easy to fix. Um, you can take things like trimethoglycine (TMG), which is an over-the-counter amino acid. Um, it's very good at fixing homocysteine metabolism. You know, one of the things that I preach about a lot is, uh, getting genetic methylation testing done. It's a test you do once in your lifetime. You look at what your, what nutrients your body can convert and what it can't convert and then you supplement for deficiency. I mean, if you want to see magic happen in human beings, you give their body the raw material it needs to do its job, right? When you deprive the human body of certain raw materials, you get the expression of disease. And then sadly what happens to most people is they say, "Well, we can't find, you know, uh, anything wrong on your labs, um, and we can't find anything wrong diagnostically with your heart. We're still going to put you on heart medication. We're still going to put you on a beta blocker, a calcium channel blocker, diuretic, ACE inhibitor. We're still going to medicate your heart even though we can't find anything wrong with it." Um, and you say, "Well, how did I get the high blood pressure?" Oh, well, your uncle has it on your mom's side and your grandfather had high blood pressure, so you have familial hypertension. You have genetically inherited hypertension. And if I can get you to believe that you have a genetically inherited disease, I can get you to subscribe to a lifetime of medication.

But if you actually just took this one step further and you ask your doctor if they tell you this, you have high, they tell you you have high blood pressure because your mom's brother has it and your father has it or your uncle had it. Um, then just look them in the eye and say, "What gene did I inherit from my ancestor that gave me this condition?" And watch their face go blank because that gene does not exist. We've mapped the entire human genome. We know every gene in the human body. If, if high blood pressure was a genetically inherited disease, we would be able to say that's the gene that you have. We don't. There are genetically inherited predispositions. The BRCA gene is a real genetic, genetic predisposition to breast cancer. But, um, what we pass from generation to generation is not disease. In fact, the human genetic, um, uh, genome is specifically designed to not pass on disease. We have mechanisms in place called S-phase arrests, cellular apoptosis, that are designed to stop disease and pathology from from progressing or we would have inherited every disease under the sun as humanity um aged over the last centuries. And so but what we do inherit is the inability for the body to refine a raw material into the usable form, which causes a deficiency, which leads to that disease. And that deficiency can be fixed. That's what's so exciting is, you know, one of the big things about um artificial intelligence, Nick, and I'm and I'm sorry if I'm eating your face.

Uh, so, you know, is that, um, the artificial intelligence is going to use voluminous amounts of data to circumvent the system. We're not going to rely on randomized clinical trials. We're not going to rely on a big pharma placebo control trial. We are going to rely on big data and big data doesn't lie. And so you're going to find that, "Wow, these were not genetically inherited diseases. These were nutrient deficiencies." When we put the, the right nutrients back in the human body, it began to metabolize homocysteine. As homocysteine began to fall, the vascular system began to relax. As the vascular system relaxed, the pressure returned to normal because there never was anything wrong with the heart. And we could go through dozens of these examples. Hypothyroid, autoimmune, Crohn's disease, Hashimoto's. Um, you know, we, we, we automatically assume when we can't explain something in medicine that it's either genetically inherited, it's familial, or it's just idiopathic. You know, so many people have autoimmune diseases and they're just led to believe, you know, if you have, uh, Crohn's that you just woke up one day and your immune system began to randomly attack your colon. We call that Crohn's. You woke up one day and it's randomly attacking your thyroid. Now you have Hashimoto's or the lacrimal gland in your eye, you have Grins, or the blood and you have Lupus, or any number of other autoimmune diseases. But, but if we just again, if we just took a step back and we said, "Well, before I accept that I woke up one day and my immune system just went haywire for no apparent reason, Um, why wouldn't I take the position that maybe God didn't make a mistake? Maybe the immune system is actually acting properly. We just need to figure out why it's there." So in other words, if, if, if this were a, this is a bottle cap, but if, if this were a mold spore or a mycotoxin or a virus or a parasite and this was a healthy cell, this doesn't hide like this. It hides like this.

That's an important distinction because the immune system is hypervigilant. It wants to get to this. It wants to go after this, this invader, right? It wants to go after this pathogen. But when it meets the cell wall, it doesn't have permission to come inside the cell.

So, you know how the immune system creates permission to kick down this cell door? It creates an antibody to that cell. And now you have an autoimmune disease. But the question is, why do you have the immune system lighting up against this specific cell? Well, maybe because this pathogen is hiding in there. The thyroid gland, for example, is, is infamous for heavy metals gravitating to the thyroid. It has an affinity for the thyroid. Um, you know, the, the immune system, 70% of our immune system is sitting right outside of our gut, right? Right outside the luminal wall of the gut is 70% of your immune system. Why? Because that's where all the action is. And so when you have that much of the immune system just sitting waiting and and contents begin to leak out of the gut, which is called leaky gut and get into the bloodstream or into the luminal area of the gut where they don't belong, the immune system is going to attack them there. And then we're going to hold the immune system responsible and say, "I'm going to give you an immunosuppressant and, uh, you know, an anti-inflammatory to control this condition," when really what we should be doing is trying to find out why the immune system lit up in the first... Some very interesting studies coming out on this now. You know, in multiple sclerosis, um, there was an autopsy study on postmortem autopsies on on multiple, uh, patients that that died with MS, um, and they found in 100% of these patients the same, uh, cestode nematode, the same helminth parasite, a, a burrowing parasite like a corkscrew parasite that had burrowed into the myelin sheath of the nerve. And so if you think about multiple sclerosis for example, um, you know, you think about a nerve as a copper wire inside of a rubber sheath, right? So if I took a knife and I nicked that copper wire, I'm sorry, nicked that rubber sheath and exposed the copper wire, as a nerve signal or electrical signals traveling down that wire, it's going to arc and it's going to jump out at that location, which is what we would call a, a sore, a sclerosis. So multiple sores, multiple scleroses in the myelin sheath of the nerve is what we call multiple sclerosis. So if the immune system were manufacturing antibodies to the myelin sheath of the nerve, why then wouldn't it uniformly dissolve that myelin sheath? Why wouldn't it be attacking all of the myelin in the body and uniformly degrading it? Why are there these bullet holes and why are there multiple scleroses, multiple sores in the myelin sheath? Because the immune system is intelligent. It is only going to burn a hole where the pathogen exists. It's not going to burn, it's not going to attack all of the myelin. It's hard. You know, even, even the, the most prominent infectious disease physicians have a hard time explaining it. I'm like, "Well, if, if this is a condition where the immune system is manufacturing an antibody to myelin, why is it selective about the myelin it attacks?" I mean, if this is this is the myelin sheath, why does it go there and then there and then here and then there? Why, why are there seem to be this random pattern to it? Well, maybe it's the cestode nematodes, maybe it's the parasitic invasion that the immune system is targeting. Why if it's manufacturing antibodies to the thyroid doesn't it attack the entire thyroid? Because maybe it's attacking the cells where the embedded mercury is. You know, why when we have Crohn's disease doesn't it destroy the whole colon? Because it's going to the area where the leaky gut is. And so I think the innate immune system is more intelligent than modern medicine. If we believed more in what God gave us and less in what man makes us, we would find the pathway to longevity because we would support these systems rather than, uh, you know, suppress them.

Oh, no question. The gut brain connections. Yeah, gut brain connection. Yes. What the gut wants, the gut gets, right? Um, and now there's evidence of two-way signaling, right? The brain driving hunger, the brain driving sugar cravings. Um, and also the microbiota, the gut, the gut bacteria driving different cravings. I mean, if you have a proliferation, um, uh, of of of C. diff, um, or you have SIBO, small intestinal bacterial overgrowth, as those bacteria proliferate, they're actually going to send signals to provide the type of food that they like to eat, which is generally high glycemic and carbohydrates.

Great question. Um, so the question is like, you know, and what's fascinating, I, I started the ultimate human platform, um, uh, two years ago because, uh, I was, uh, a co-founder of one of the largest functional medicine clinics in the country and I realized that almost regardless of age and and gender, um, or socioeconomic status, I, I realized that almost 100% of the patients when they would come into the clinic had the exact, exact same question. Um, and and it was, you know, where do I start? Like I've been online, I, I saw NMN, NAD, res... Now I heard I should be microdosing with mushrooms. Um, and all those things have a place. Um, so the majority of people are supplementing for the sake of supplementing. Yes. They are not supplementing for deficiency. So the first thing that you need to do is you need to get data because just like, um, you know, in my, you know, a palm tree leaf rotting example, you know, you have a palm tree, you have a leaf rotting in the palm tree, if you didn't core test the soil and find out that it didn't have nitrogen and add nitrogen to the soil, nothing else would have mattered, right? Water is great for plants. If you watered the soil, the leaf would still be dying. If you put sulfur on the soil, which is great for plants, the leaf would still be dying. I put phosphorus on the soil, the leaf would still be dying. And all of those are great for plants. Nitrogen is what that plant was deficient in. Human beings are no different. So the way that you find nutrient deficiencies is twofold. Number one, I believe, um, and I'm desperately trying to get this test to Australia right now. Um, and if you know anyone that can help me, I'd love to talk to him. Um, you know, just going to try to work through the regulatory regime to get our, our, our DNA testing because it's been very successful in the rest...

Great. Um, then we're going to, we're going to change, uh, you know, Australia's future because I believe so much in in in just looking at a very specific class of genes called the genes of methylation, meaning what can your body convert into the usable form and what can it not? And you start by supplementing with that deficiency, right? Because if you don't find that deficiency, almost nothing else matters. If you have the MTHFR gene mutation, you don't know it and you're eating a healthy diet of folate, which is in green leafy vegetables, or you're supplementing with folic acid, that is useless. You are going to be deficient in a key methylated nutrient called methylfolate. And if you don't know it, it that is so far downstream, or sorry, upstream in the methylation cycle that you're going to have a strange myriad of symptoms that you'll never be able to put a finger on. Anxiety, ADD, ADHD. You're going to be one of those people that lays down to go to sleep at night and your body tired but your mind awake.

Easy to fix. I'm, I'm going to bring some of these, uh, supplements and maybe a few of these tests with me. Um, and and, uh, I'm, I'm hosting an event, uh, in a live audience. So hope, hopefully Australia doesn't cuff me from bringing some methyl...

Okay. Um, then, uh, uh, and and we're going to talk about, you know, starting with a genetic methylation test, right? And what this will say is, "You are deficient in in in the following nutrients." And there will never be a time in your b, in your life where you magically um can methylate these because your genes, you're born with, are the genes you die with. And that's where you start. You start with um supplementing for the basics. It usually is a complex of B vitamins, a specific form of B12 called methylcobalamin, um, very often, uh, methylfolate, uh, trimethoglycine, um, for homocysteine metabolism and then sometimes for mood and emotional states, uh, something called SAM-e. That's where you start. And you, you put the nitrogen back into the soil and now you move to the next step, right? How do I improve, how do I go from being, you know, good to better to best to maybe even being optimal? And so you start with supplementing for deficiency. That's an exact road map. Um, it's a science. There's not a guessing game to it. Or else you get stuck in the paralysis of analysis. Should I be on nicotinamide riboside, nicotinic acid, nicotinamide mononucleotide, or should I take NAD directly? Should I do it by IV? Should I do it by injection? I thought you couldn't take it orally, but then somebody has a liposome, I can take it orally. It's, I mean, you, you will drive yourself crazy. I do this for a living and it still drives me crazy. Um, and so if the, if the body doesn't have the basic raw materials it needs to do its job, nothing else matters. And then the next step that you, um, do is, is you really look at your lifestyle and you ask yourself, um, you know, am, am I living a healthy lifestyle? Am I living a life, a longevity lifestyle if that's your goal? Um, am I moving a lot? Do I prioritize sleep? Sleep is our human superpower. You cannot diet your way around poor sleep. You can't exercise your way out of poor sleep. You can't exercise your way around a poor diet. So there's some foundational, um, you know, basics. Supplement for deficiency. Optimize your sleep. Um, and transition to a whole food diet and make exercise non-negotiable. You don't have to be a gym rat. You don't have to be a CrossFit athlete. You don't have to learn compound Olympic lifts. You have to just move. Walking is probably the most underrated exercise in the world. And and the reason for that is, um, we know that walking is good for our blood sugar. We know that walking is good to lose weight. But what we don't realize is that walking is integral to the movement of lymphatic fluid. And our lymph system, which is the system that we use to eliminate waste from the body, is a static system. It doesn't have the pressure of the heart behind it, right? Our arteries and veins are going to circulate because of the ventricular contraction of our heart. Your lymphatic system is static. This is why people in hospitals get septic. People that are bedridden very often die of septic infections because the lymph is not moving. It moves by muscular contraction, right? So, um, just the simple act of walking. God designed to perfectly move our lymphatic system. This is why Tai Chi, yoga has such profound impacts on your health. Not only do they increase your flexibility and increase your strength, which is amazing, but the majority of the impact is coming from the fact that they are eliminating toxins and waste. You have a lymphatic system in your brain. It's called the glymphatic system. This system is only active during deep sleep. So when people say, "Why is sleep so important? Why do I feel so good after a good night's sleep?" Because during the deep phases of sleep, you've actually activated the glymphatic system. Your brain actually dwells a little bit. You swell, your, your brain is actually slightly swollen during the day and these, um, these channels are actually closed. At night during deep sleep, the brain loses, um, some size, some mass and and these channels open. And now the brain eliminates waste. It repairs, it detoxifies and regenerates. If you're not sleeping, your brain is not detoxifying, full stop. Now there are other stages of sleep like REM sleep where we're assembling memory. We're taking the prefrontal cortex and conscious thought and learned activities of the day and we're marrying those with the hippocampus and creating learned memory. So if you're a student and you're paying attention to a lecture or if you're an entrepreneur and you're working on your business plan or if you're just going through life and you want to have good short-term recall, healthy focus, um, uh, healthy waking energy, then REM stages of sleep are critical to that longevity profile. So movement, whole food diet, supplementing for deficiency and targeting your sleep. Those are the fastest ways to completely turn your life around if you're in a place where, you know, you're just not feeling so well or doing that well. And then you start to stack on the the really interesting stuff, the modalities, um, that are, you know, proven to not only improve function now but to extend life. Um, you know, saunas are probably the most incredible way to, um, as a single modality, um, to ward off all cause mortality, to reduce cardiovascular disease, um, disease risk.

I love an ice. These hormetic stresses, you know, I have a saying that aging is the aggressive pursuit of comfort. You know, one of the reasons why we're aging so fast is because we aggressively seek comfort. To stop telling grandma not to go outside, it's too hot. Not to go outside, it's too cold. Just to lay down, just to relax. To just eat at the first pang of hunger, right? Because this is collapsing her natural defense mechanisms. Look, I don't care what side of the aisle you're on. The worst thing that happened to humanity during the pandemic was residential quarantining, masking and social distancing. And that's started with a...

Unbelievable. Unbelievable crime against humanity. What happened there? And it happened here too. Look, um, but the reason for that is not just because of the skyrocketing rates of alcoholism, the, the, the, the people weren't getting adequate medical treatment, they, the cancers went misdiagnosed, you know, undiagnosed. Um, you know, uh, but, uh, and the, and the social ostracization, the isolation, which I could, we could go way down the road...

The issue was that human beings were designed to be in the proximity of other human beings.

And there's something called hormesis, which is an applied stress to the body where the body strengthens, right? Hormetic stress. So cold, cold exposure is a hormetic stress. Heat is a hormetic stress. So when the body is stressed, it strengthens. If you don't load a bone, it never strengthens. If you don't actually tear a muscle, it doesn't grow. If you don't challenge the immune system, it weakens. And so we did the opposite of what we wanted to do. We then entered the pandemic of a globally weakened immune system. And what is the consequences of a globally weakened immune system? Well, now you start to hear about all these things. Monkeypox. "Well, what the hell is Monkeypox?" Nobody ever heard of Monkeypox. "Well, everybody's dying of it now." Well, because it's such a weak virus that it was actually able to proliferate, um, because of the weakened, the pandemic of weakened immune systems. And now you're seeing the consequences of that. I'm not even talking about all the psychiatric implications and what happened in the schools and what happened to collegiate kids and people that lost their athletic careers because the, the formidable years of their life have gone by. But the, the weakening of the immune system. So, um, so I would begin to engage in activities that create hormetic stress. Red light therapy is incredible. Um, full body red light therapy. Um, saunas, um, cold plunging is one of the most phenomenal um, you know, I wouldn't say discoveries because we've been doing it for centuries, but phenomenal trends of of this century. You know, when you, when you immerse yourself in cold water, um, because water is 29 times more thermogenic than air, this, this cascade of of incredibly beneficial things happen. You get a peripheral vasoconstriction, um, which drives blood to the brain, to the heart, and to the lungs. And by the way, in that order, um, and and then all the the vital organs, you get a release of something called cold shock proteins. If you really, really want to have some fun sometime, Google around about cold shock proteins. These are, we're only now marginally beginning to understand these. Um, there are certain cold shock proteins like lin 28A and lin 28B that are being studied for um their direct impact on improving insulin resistance. So increasing insulin sensitivity. You want to be sensitive to insulin. You don't want to be resistant to insulin. Um, they scour the body of free radical oxidation. They improve the rate of protein synthesis if done before exercise, which is muscle repair. Um, you know, you, you get a you get a massive release of dopamine. Um, uh, which improves your alertness. It elevates your mood. It elevates your emotional state. And then maybe most profoundly, you get an activation of something called brown fat, which is a very special type of fat in the body that exchanges a calorie for a measure of heat. Um, and anybody that tells you that cold plunging doesn't burn fat has just not read the science. I get attacked for it all the time online because I say nothing for a period of time will do more to burn fat in your body than cold plunging because there is a cost to warming up. If you got in a cold plunge, let's say 50 degrees Fahrenheit and then you get out and you're in a room that's 70 degrees Fahrenheit, your body's going to go back to 98.6 degrees Fahrenheit. So how is it right now that you as you sit there are warmer than the room you're in? There's a cost to that and the cost is calories. We actually take a calorie and we turn it into a measure of heat. In fact, the definition of a calorie is the amount of heat that it takes to raise one cubic centimeter of water 1° centigrade. So if we measure calories in heat and we're heat deficient and we create heat to return to that gradient, then obviously we're burning a voluminous amount of calories. So you get all of that from three minutes of cold exposure. The problem with cold plunging is just like every other biohack is that we take it to the extremes. We automatically think colder is better, longer is better. You know, I see people getting in near freezing water with a straw, I'm sorry, with a snorkel and going underwater and staying underwater for 12 minutes. That is horrible for you.

It's your brain is only this far inside the surface of your skull, Nick. It's not good to freeze it. It's not good to bake it.

Thank you so much, brother. I look forward to to meeting you in Australia and uh any help you can give me on getting the genetic test and supplements into your country. I I I really love the Australian people. I've got a lot of followers, a lot of members of my VIP community are Australian. Um so I can't wait to get to the land down under and and meet you face to face.

Thank you.